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Color Plates

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Figures

Image of Color Plate 1.

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Color Plate 1.

(A) Fluorescently labeled strain B31 visualized by fluorescent microscopy (magnification, ×250). (B) Picture of a semi-engorged attached female adult tick (provided by K. A. de Groot).

Citation: Fratamico P, Smith J, Brogden K. 2009. Color Plates, In Sequelae and Long-Term Consequences of Infectious Diseases. ASM Press, Washington, DC.
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Color Plate 2.

Different organs can be affected during the course of human Lyme borreliosis. (A) Picture of a patient with left-sided antibiotic-refractory Lyme arthritis. (This picture was kindly provided by A. C. Steere.) (B) Picture of a patient with a right-sided nervus facialis paresis. This is usually a sign of early neuroborreliosis; however, in a minority of cases this condition does not totally recover, causing a chronic disability. (The picture was a kind gift from P. Speelman and J. de Gans.) (C) Unilateral acrodermatitis chronica atrophicans in two European patients. (These pictures were kindly provided by D. J. Tazelaar.) (D) Holter registration of a total atrioventricular block in a patient with Lyme carditis (with an onset several years after the tick bite). (This picture was kindly provided by J. Verbunt.)

Citation: Fratamico P, Smith J, Brogden K. 2009. Color Plates, In Sequelae and Long-Term Consequences of Infectious Diseases. ASM Press, Washington, DC.
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Color Plate 3.

Rheumatic stenotic mitral valve status postring repair with moderate fibrosis and large vegetation (VGS) with anterior leaflet perforation. (Courtesy of Louis R. Dibernardo.)

Citation: Fratamico P, Smith J, Brogden K. 2009. Color Plates, In Sequelae and Long-Term Consequences of Infectious Diseases. ASM Press, Washington, DC.
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Color Plate 4.

Poststreptococcal AGN. (Courtesy of David Howell.)

Citation: Fratamico P, Smith J, Brogden K. 2009. Color Plates, In Sequelae and Long-Term Consequences of Infectious Diseases. ASM Press, Washington, DC.
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Color Plate 5.

Section of an endometrial aspirate showing intensive infiltration with plasma cells scattered over the mucosa, a finding which can be seen with both gonococcal and chlamydial endometritis.

Citation: Fratamico P, Smith J, Brogden K. 2009. Color Plates, In Sequelae and Long-Term Consequences of Infectious Diseases. ASM Press, Washington, DC.
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Color Plate 6.

Total destruction of fallopian tube epithelium after an episode of salpingitis.

Citation: Fratamico P, Smith J, Brogden K. 2009. Color Plates, In Sequelae and Long-Term Consequences of Infectious Diseases. ASM Press, Washington, DC.
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Color Plate 7.

PAS-positive macrophages in lamina propria.

Citation: Fratamico P, Smith J, Brogden K. 2009. Color Plates, In Sequelae and Long-Term Consequences of Infectious Diseases. ASM Press, Washington, DC.
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Color Plate 8.

Malaria life cycle correlated to numbers of parasites at each stage. While the clinical manifestations of malaria correlate with the blood stages, the infection begins when a female mosquito injects hundreds of sporozoites into the dermis and only one or two invade hepatocytes. In a week, 5 to 10,000 progeny called merozoites burst from the single hepatocyte to start the cycle of erythrocyte invasion. In the case of which multiplies at a rate of tenfold every 2 days, soon billions of parasites are circulating. Less than 0.1% differentiates into male and female gametocytes. The female mosquito again bites to imbibe 1 to 3 microliters of blood, containing 10 to 100 gametocytes. After fertilization, the zygote transforms into a ookinete, which penetrates through the stomach to the basement membrane. Only one or two oocysts develop per infectious bite. Each oocyst will produce thousands of sporozoites, which migrate to the salivary glands, ready for the cycle to begin again.

Citation: Fratamico P, Smith J, Brogden K. 2009. Color Plates, In Sequelae and Long-Term Consequences of Infectious Diseases. ASM Press, Washington, DC.
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Color Plate 9.

Differentiation of human species. The four principal human malaria species can be separated on the basis of blood film examinations and the chronology of the different stages as well as the ability to be dormant in the liver to cause relapse months later, in the case of .

Citation: Fratamico P, Smith J, Brogden K. 2009. Color Plates, In Sequelae and Long-Term Consequences of Infectious Diseases. ASM Press, Washington, DC.
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Color Plate 10.

Two cases of post-kala-azar dermal leishmaniasis from Sudan, showing nodular eruption, with occasional confluence of the lesions. (Photographs courtesy of J. Dereure, with permission.)

Citation: Fratamico P, Smith J, Brogden K. 2009. Color Plates, In Sequelae and Long-Term Consequences of Infectious Diseases. ASM Press, Washington, DC.
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Image of Color Plate 11.

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Color Plate 11.

Scar patterns following the cures of various localized cutaneous leishmaniasis. (A) Multiple scars of an CL from Peru. (B) Large whitish scar following a CL from Morocco (photograph courtesy of J. Dereure, with permission). (C) Hyperpigmented scar of an CL from Senegal. (D and E) Reactivation of scars in CL from French Guiana (photograph in panel D courtesy of P. Couppié, with permission, and photograph in panel E courtesy of E.R. Pradinaud, with permission).

Citation: Fratamico P, Smith J, Brogden K. 2009. Color Plates, In Sequelae and Long-Term Consequences of Infectious Diseases. ASM Press, Washington, DC.
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Image of Color Plate 12.

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Color Plate 12.

Mucosal lesions of MCL cases from Peru. The first granulomatous lesion of the anterior part of nasal septum (A) is rapidly destroying the nasal septum (B and C). (D) Pharyngeal granulomatous lesions. (E) External appearance of the nasal pyramid. (Photographs in panels A, B, C, and D courtesy of A. Llanos-Cuentas, with permission, and photograph in panel E courtesy of J.P. Guthmann, with permission.)

Citation: Fratamico P, Smith J, Brogden K. 2009. Color Plates, In Sequelae and Long-Term Consequences of Infectious Diseases. ASM Press, Washington, DC.
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Color Plate 13.

The mucosal lesions (A) and their subsequent sequelae, with retractive scars (B), are generally highly disfiguring, which has considerable socio-psychological impact for the patient. (Photographs courtesy of A. Llanos-Cuentas, with permission.)

Citation: Fratamico P, Smith J, Brogden K. 2009. Color Plates, In Sequelae and Long-Term Consequences of Infectious Diseases. ASM Press, Washington, DC.
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Color Plate 14.

Necropsy aspect of a chronic Chagas cardiomyopathy. (A) External aspect with considerable dilatation of the heart. The small protrusion of the apex (arrow) corresponds to a ventricular apical aneurysm. (B) At the site of the aneurism, myocardium has disappeared, and only a thin membrane remains, consisting of pericardium and endocardium closely joined, as shown by transillumination.

Citation: Fratamico P, Smith J, Brogden K. 2009. Color Plates, In Sequelae and Long-Term Consequences of Infectious Diseases. ASM Press, Washington, DC.
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Color Plate 15.

Photomicrographs of lung from an immunosuppressed patient who developed opportunistic infection by CMV. (A) Widening of the interstitium and occasional enlarged alveolar epithelial lining cells. Magnification, ×40. (B) The characteristic “owl’s eye” intranuclear CMV inclusion in the cell at center (hematoxylin and eosin stains). Magnification, ×200.

Citation: Fratamico P, Smith J, Brogden K. 2009. Color Plates, In Sequelae and Long-Term Consequences of Infectious Diseases. ASM Press, Washington, DC.
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Color Plate 16.

Histological preparations of adenovirus pneumonia in a pediatric patient. (A) Section stained with hematoxylin and eosin showing bronchial lumen partially filled with smudge cells. (B) In situ hybridization with an adenovirus probe of a serial section of the lung shown in panel A showing virus-positive epithelial cells lining the bronchioles (arrowheads) as well as in the parenchyma (arrows). Original magnification, ×250 (79).

Citation: Fratamico P, Smith J, Brogden K. 2009. Color Plates, In Sequelae and Long-Term Consequences of Infectious Diseases. ASM Press, Washington, DC.
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Image of Color Plate 17.

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Color Plate 17.

Immunohistochemical staining of a formalin-fixed paraffin-embedded section of human lung tissue with an antibody to adenovirus E1A. Arrows indicate alveolar epithelial cells that express adenovirus E1A protein. (Courtesy of Mark Elliott.)

Citation: Fratamico P, Smith J, Brogden K. 2009. Color Plates, In Sequelae and Long-Term Consequences of Infectious Diseases. ASM Press, Washington, DC.
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